Search results for "Prostaglandin E2"

showing 10 items of 63 documents

Tumor-Derived Prostaglandin E2 Promotes p50 NF-κB-Dependent Differentiation of Monocytic MDSCs

2020

Abstract Myeloid-derived suppressor cells (MDSC) include immature monocytic (M-MDSC) and granulocytic (PMN-MDSC) cells that share the ability to suppress adaptive immunity and to hinder the effectiveness of anticancer treatments. Of note, in response to IFNγ, M-MDSCs release the tumor-promoting and immunosuppressive molecule nitric oxide (NO), whereas macrophages largely express antitumor properties. Investigating these opposing activities, we found that tumor-derived prostaglandin E2 (PGE2) induces nuclear accumulation of p50 NF-κB in M-MDSCs, diverting their response to IFNγ toward NO-mediated immunosuppression and reducing TNFα expression. At the genome level, p50 NF-κB promoted binding …

0301 basic medicineCancer ResearchCellular differentiationProstaglandin E2 receptormedicine.medical_treatmentMelanoma ExperimentalApoptosisSettore MED/08 - Anatomia PatologicaNitric OxideDinoprostoneMonocytesInterferon-gammaMice03 medical and health sciences0302 clinical medicineImmune systemOxytocicsImmune ToleranceTumor Cells CulturedmedicineAnimalsHumansProstaglandin E2Cell ProliferationChemistryMyeloid-Derived Suppressor CellsNF-kappa B p50 SubunitCell DifferentiationImmunotherapyAcquired immune systemPancreatic Neoplasms030104 developmental biologyOncologyp50 NF-κB differentiation of monocytic MDSC.030220 oncology & carcinogenesisMyeloid-derived Suppressor CellCancer researchTumor necrosis factor alphaColorectal Neoplasmsmedicine.drugCancer Research
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Enzymatic Activity of HPGD in Treg Cells Suppresses Tconv Cells to Maintain Adipose Tissue Homeostasis and Prevent Metabolic Dysfunction.

2019

Summary Regulatory T cells (Treg cells) are important for preventing autoimmunity and maintaining tissue homeostasis, but whether Treg cells can adopt tissue- or immune-context-specific suppressive mechanisms is unclear. Here, we found that the enzyme hydroxyprostaglandin dehydrogenase (HPGD), which catabolizes prostaglandin E2 (PGE2) into the metabolite 15-keto PGE2, was highly expressed in Treg cells, particularly those in visceral adipose tissue (VAT). Nuclear receptor peroxisome proliferator-activated receptor-γ (PPARγ)-induced HPGD expression in VAT Treg cells, and consequential Treg-cell-mediated generation of 15-keto PGE2 suppressed conventional T cell activation and proliferation. C…

0301 basic medicineanalogs & derivatives [Dinoprostone]Malemetabolism [Diabetes Mellitus Type 2]Adipose tissueLymphocyte Activation15-ketoprostaglandin E2T-Lymphocytes RegulatoryJurkat cellsJurkat CellsMice0302 clinical medicineimmunology [Lymphocyte Activation]genetics [Insulin Resistance]STAT5 Transcription FactorHomeostasisImmunology and AllergyTissue homeostasisgenetics [Hydroxyprostaglandin Dehydrogenases]Mice Knockoutcytology [Intra-Abdominal Fat]enzymology [T-Lymphocytes Regulatory]FOXP3hemic and immune systems3T3 CellsCell biologyInfectious Diseases030220 oncology & carcinogenesisHydroxyprostaglandin Dehydrogenasesmedicine.symptomimmunology [T-Lymphocytes Regulatory]metabolism [STAT5 Transcription Factor]Immunologymetabolism [Dinoprostone]chemical and pharmacologic phenomenaInflammationIntra-Abdominal FatBiologyDinoprostoneCell Line03 medical and health sciencesmetabolism [Hydroxyprostaglandin Dehydrogenases]immunology [Homeostasis]medicineAnimalsHumansddc:610immunology [Intra-Abdominal Fat]HEK 293 cells030104 developmental biologyHEK293 CellsDiabetes Mellitus Type 2Cell cultureInsulin ResistanceHomeostasis
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Participation of Heme Oxygenase-1 in a Model of Acute Inflammation

2003

In this study, the role of heme oxygenase-1 (HO-1) in the inflammatory response elicited by zymosan in the mouse air pouch model has been examined. This response is characterized by a time-dependent increase in HO-1 expression in the leukocytes migrating into the exudates. At 24–48 h maximal HO-1 expression was accompanied by reduced cyclooxygenase-2 (COX-2) and nitric oxide synthase-2 (NOS-2) expression as well as low levels of inflammatory mediators. Hemin administration into the air pouch caused an elevation of HO-1 protein and bilirubin levels induced by zymosan with inhibition of COX-2 expression. In mouse peritoneal macrophages from hemin-injected mice, we also observed an increased …

0301 basic medicinemedicine.medical_specialtyNitric Oxide Synthase Type IIInflammationGeneral Biochemistry Genetics and Molecular BiologyNitric oxideMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicinemedicineAnimalsProstaglandin E2HemeInflammationbiologyZymosanZymosanMembrane ProteinsIsoenzymesHeme oxygenaseDisease Models Animal030104 developmental biologyEndocrinologychemistryCyclooxygenase 2Prostaglandin-Endoperoxide Synthases030220 oncology & carcinogenesisHeme Oxygenase (Decyclizing)ImmunologyMacrophages Peritonealbiology.proteinHeminFemaleCyclooxygenaseNitric Oxide Synthasemedicine.symptomHeme Oxygenase-1medicine.drugHeminExperimental Biology and Medicine
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Tumor-derived immuno-modulators induce overlapping pro-tolerogenic gene expression signatures in human dendritic cells.

2016

Immature dendritic cells (iDCs) and tolerogenic DCs are essential for the induction and maintenance of peripheral tolerance. Tumors produce immuno-modulatory factors which imprint a pro-tolerogenic, maturation-resistant state in DCs. Here we asked for common markers of differentially tolerized human monocyte-derived DC populations. For this, PBMC-derived monocytes were differentiated to DCs in the presence of established immuno-modulators as released by tumors (IL-6, IL-10, TGF-β, glucocorticoid [GC], prostaglandin E2 [PGE2]). Most unstimulated pro-tolerogenic DC populations commonly over-expressed some tolerance-associated markers (ILT-4, IL-10, HO-1) as compared with iDCs. These markers m…

0301 basic medicinemedicine.medical_treatmentT cellT-LymphocytesImmunologyStimulationBiologyLymphocyte ActivationDinoprostone03 medical and health sciences0302 clinical medicineDownregulation and upregulationNeoplasmsmedicineImmune ToleranceImmunology and AllergyHumansImmunologic FactorsProstaglandin E2GlucocorticoidsCells CulturedAntigen PresentationPeripheral toleranceCell DifferentiationGeneral MedicineDendritic CellsInterleukin 10030104 developmental biologyCytokinemedicine.anatomical_structureImmunologyB7-1 AntigenCytokinesCD80Heme Oxygenase-1030215 immunologymedicine.drugHuman immunology
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β-Adrenoceptor stimulation up-regulates phosphodiesterase 4 activity and reduces prostaglandin E 2 -inhibitory effects in human neutrophils

2000

Human neutrophils were treated for 4 h with a combination of salbutamol (1 µM), a β2-adrenoceptor agonist, and rolipram (30 µM), a selective phosphodiesterase 4 inhibitor, to investigate whether this treatment produces up-regulation of phosphodiesterase activity with functional consequences. Anion-exchange chromatography coupled with the use of selective activators and inhibitors demonstrated that a phosphodiesterase activity with characteristics of the isoenzyme type 4 was increased in drug-treated cells. Kinetic analysis showed a ~1.5-fold increase in V max without alteration of K m values. The augmented phosphodiesterase activity in drug-treated cells was abolished by actinomycin D. Cycl…

AdultAgonistmedicine.medical_specialtyNeutrophilsmedicine.drug_classStimulationIn Vitro TechniquesBiologyDinoprostoneNeutrophil Activationchemistry.chemical_compoundPDE4BDownregulation and upregulationSuperoxidesInternal medicineCyclic AMPmedicineHumansAlbuterolRNA MessengerEnzyme InhibitorsProstaglandin E2RolipramPharmacologyReverse Transcriptase Polymerase Chain ReactionSuperoxideZymosanZymosanGeneral MedicineAdrenergic AgonistsCyclic Nucleotide Phosphodiesterases Type 4Up-RegulationEndocrinologychemistry3'5'-Cyclic-AMP PhosphodiesterasesReceptors Adrenergic beta-2Roliprammedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Role of prostaglandin E2 in the invasiveness, growth and protection of cancer cells in malignant pleuritis.

2006

The recurrence of pleural effusions is a common event in a variety of neoplastic diseases. The objective of this study was to identify the mechanisms promoting the homing and growth of cancer cells within the pleural space. A cancer cell line recovered from malignant pleural fluids (lung adenocarcinoma cell line) that constitutively expresses cyclooxygenase 2 (COX-2) and all types of prostaglandin receptors was studied. It was first demonstrated using a matrigel system, that malignant pleural fluids increase the invasiveness of adenocarcinoma cells more than congestive heart failure (CHF) pleural fluids. Moreover, exposure to exudative malignant, but not to CHF pleural fluids, increased the…

AdultCancer ResearchPathologymedicine.medical_specialtyLung NeoplasmsPleural effusionAdenocarcinomaDinoprostoneMetastasisCell Line TumormedicineHumansReceptors Prostaglandin ENeoplasm InvasivenessProstaglandin E2PleurisyAgedCell ProliferationMatrigelCyclooxygenase 2 Inhibitorsbusiness.industryNF-kappa BCancerMiddle Agedmedicine.diseaseNeoplasm ProteinsPleural Effusion MalignantUp-RegulationOncologyPleurisyCyclooxygenase 2TalcCancer cellAdenocarcinomabusinessmedicine.drugEuropean journal of cancer (Oxford, England : 1990)
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Effect of loperamide on jejunal electrolyte and water transport, prostaglandin E 2-induced secretion and intestinal transit time in man

1991

Jejunal perfusion was performed in 12 healthy volunteers to evaluate the dose dependent effects of loperamide on intestinal absorption, stimulated secretion and transit. In 6 volunteers intestinal perfusion of the jejunal segment with isotonic NaCl solution was followed by addition of loperamide in increasing doses (2–8 mg·l−1). The volunteers were pretreated with 1 mg·l−1 prostaglandin E2 (PgE2) in the perfusate before addition of 4 mg·l−1 loperamide. Phenolsulphonphtalein (PSP) boluses (2 ml) were given to measure mean transit time (MTT). Loperamide 2 mg·l−1 converted the minor secretion after perfusion with the standard solution (water −1.45 ml·min−1, Na −0.09 and Cl −0.04 mmol·min−1) to…

AdultDiarrheaMalemedicine.medical_specialtyLoperamideAdolescentAbsorption (skin)LoperamideDinoprostoneIntestinal absorptionJejunumChloridesInternal medicinemedicineHumansPharmacology (medical)Prostaglandin E2Gastrointestinal TransitPharmacologyWater transportDose-Response Relationship DrugChemistrySodiumBiological TransportGeneral MedicineWater-Electrolyte BalanceJejunumEndocrinologymedicine.anatomical_structureIntestinal AbsorptionMechanism of actionmedicine.symptomPerfusionmedicine.drugEuropean Journal of Clinical Pharmacology
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Increased prostaglandin E2 concentrations and cyclooxygenase-2 expression in asthmatic subjects with sputum eosinophilia.

2003

Abstract Background Prostaglandin E 2 (PGE 2 ) is known to be produced within human airways, but it is not clear whether in airway diseases it can play a deleterious or a beneficial role. Recently it has been reported that PGE 2 can enhance eosinophil survival in vitro. Objective To evaluate whether the concentrations of PGE 2 in asthmatic airways correlate with the number of eosinophils and can be responsible for eosinophil-enhanced survival and to identify the cyclooxygenase isoform contributing to the synthesis of PGE 2 by cells present in asthmatic airways. Methods Reversed-phase high-performance liquid chromatography and/or specific radioimmunoassay was used to measure PGE 2 concentrat…

AdultMaleSputum Cytologymedicine.medical_treatmentImmunologyApoptosisDinoprostoneLeukocyte CountRibonucleasesEosinophiliaImmunology and AllergyMedicineHumansProstaglandin E2Eosinophil cationic proteinbiologybusiness.industryEosinophil Granule ProteinsOsmolar ConcentrationSputumMembrane ProteinsBlood ProteinsEosinophilEosinophil Granule ProteinsMiddle AgedImmunohistochemistryAsthmaEosinophilsIsoenzymesmedicine.anatomical_structureCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesImmunologybiology.proteinSputumFemaleCyclooxygenasemedicine.symptombusinessmedicine.drugProstaglandin EThe Journal of allergy and clinical immunology
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The exposure of healthy volunteers to 200 ppm 1,1,1-trichloroethane increases the concentration of proinflammatory cytokines in nasal secretions

1999

Objectives: Irritating effects of organic solvents have usually been measured by means of questionnaires. The aim of the present study was to evaluate the sensitivity of different methods of detecting subclinical irritating effects. Methods: Twelve healthy, non-smoking students were exposed to 200 ppm and to 20 ppm 1,1,1-trichloroethane in an exposure chamber, using a crossover design. The amounts of interleukins (IL)-1β, IL-6 and IL-8 and prostaglandin E2 (PGE2) in nasal secretions were measured. Mucociliary transport time was determined with the saccharine test. Ciliary beat frequency of nasal epithelial cells was measured with video-interference contrast microscopy. Subjective symptoms w…

AdultMalemedicine.medical_specialtyMucous membrane of nosemedicine.disease_causeDinoprostoneStatistics NonparametricProinflammatory cytokineInternal medicinemedicineHumansTrichloroethanesProstaglandin E2Subclinical infectionCross-Over StudiesInhalationInterleukin-6ChemistryInterleukinsInterleukin-8Public Health Environmental and Occupational HealthCrossover studyNasal MucosaEndocrinologyMucociliary ClearanceImmunologyToxicitySolventsIrritationInterleukin-1medicine.drugInternational Archives of Occupational and Environmental Health
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Prostaglandin E2 activates the ciliary beat frequency of cultured human nasal mucosa via the second messenger cyclic adenosine monophosphate.

2001

Prostaglandins influence the ciliary beat frequency (CBF) of ciliated nasal epithelial cells and a stimulatory effect has been described for prostaglandin E2 (PGE2). Until now, it is not known whether PGE2 has direct ciliostimulatory properties or acts through a second messenger. In this study we investigated whether cyclic adenosine monophosphate (cAMP) is implicated in the signal transduction pathway of PGE2-induced activation of CBF. Ciliated cells of the nasal mucosa were cultured for up to 5 days whereafter the culture medium was removed and the cells were incubated with different concentrations of test solutions. The ciliated cells were recorded under a phase-contrast microscope and v…

AdultMalemedicine.medical_specialtyStimulationMucous membrane of noseBiologyIn Vitro TechniquesSecond Messenger SystemsDinoprostonechemistry.chemical_compoundInternal medicinemedicineCyclic AMPHumansCyclic adenosine monophosphateCiliaProstaglandin E2Cells CulturedAgedDose-Response Relationship DrugColforsinEpithelial CellsGeneral MedicineMiddle AgedEpitheliumCell biologyNasal Mucosamedicine.anatomical_structureEndocrinologyOtorhinolaryngologychemistryCell cultureSecond messenger systemFemaleSignal transductionmedicine.drugSignal TransductionEuropean archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology - Head and Neck Surgery
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